Author

N. L. Miller

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Microbiology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Assay:structural biology· 2 Organism:virus· 2 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Organism:human· 1 Assay:protein structure· 1

Frequent Co-Authors

R. Raman· 2 shared R. Sasisekharan· 2 shared T. Clark· 2 shared C. M. Vezina· 1 shared E. Ricke· 1 shared M. M. Grabowska· 1 shared M. Ramirez-Solano· 1 shared P. Popovics· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersMicrobiology

R. Raman

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

A structural dynamic explanation for observed escape of SARS-CoV-2 BA.2 variant mutation S371L/F

N. L. Miller, T. Clark, R. Raman, R. Sasisekharan

The paper explains the broad immune escape of SARS-CoV-2 BA.2 variant mutations S371L/F through structural analysis of spike trimer conformational dynamics.

bioRxiv · Feb 25, 2022 · PreprintMicrobiology

Mapping Potential Antigenic Drift Sites (PADS) on SARS-CoV-2 Spike in Continuous Epitope-Paratope Space

N. L. Miller, T. Clark, R. Raman, R. Sasisekharan

The paper identifies potential antigenic drift sites on SARS-CoV-2 spike protein and projects them into epitope-paratope space to assess mutation synergy across variants of concern.

bioRxiv · Jun 7, 2021 · PreprintImmunology

Prostatic osteopontin expression is associated with symptomatic benign prostatic hyperplasia

P. Popovics, W. N. Awadallah, S. Kohrt, T. C. Case, N. L. Miller, E. Ricke, W. Huang, M. Ramirez-Solano, Q. Liu, C. M. Vezina, R. J. Matusik, W. A. Ricke, M. M. Grabowska

This study shows that osteopontin (OPN) is more abundant in symptomatic benign prostatic hyperplasia (S-BPH) compared to incidental BPH, and OPN secretion is stimulated by pro-inflammatory cytokines, driving pro-inflammatory gene expression in stromal cells.

bioRxiv · Dec 23, 2019Pathology