Author

R. Kamm

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Bioengineering· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 3 Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Approach:mechanistic / dynamical model· 1

Frequent Co-Authors

C. Hajal· 2 shared G. Offeddu· 2 shared L. Ibrahim· 2 shared Z. Wan· 2 shared A. Khalil· 1 shared A. Richards· 1 shared C. Foley· 1 shared D. Mooney· 1 shared

Activity by Year

2018-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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2 shared papersCancer Biology

C. Hajal

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

SARS-CoV-2 infection of human pluripotent stem cell-derived vascular cells reveals smooth muscle cells as key mediators of vascular pathology during infection

A. Richards, A. Khalil, M. Friesen, T. Whitfield, T. Lungjangwa, L. Gehrke, Z. Wan, X. Gao, R. Kamm, D. Mooney, R. Jaenisch

This study infects human pluripotent stem cell-derived vascular cells with SARS-CoV-2 and finds that smooth muscle cells drive vascular inflammation and coagulation.

bioRxiv · Aug 6, 2023 · PreprintMicrobiology

The effects of luminal and trans-endothelial fluid flows on the extravasation and tissue invasion of tumor cells in a 3D in vitro microvascular platform

C. Hajal, L. Ibrahim, J. C. Serrano, G. Offeddu, R. Kamm

This study examines how different fluid flows affect tumor cell extravasation and invasion in a 3D in vitro model, finding that luminal and trans-endothelial flows enhance these processes.

bioRxiv · Sep 23, 2020Cancer Biology

Glycocalyx-Mediated Vascular Dissemination of Circulating Tumor Cells

G. Offeddu, C. Hajal, C. Foley, Z. Wan, L. Ibrahim, M. Coughlin, R. Kamm

This study shows tumor cells use glycocalyx components to adhere to endothelium and invade stroma, suggesting targeting this pathway could reduce metastasis.

bioRxiv · Apr 28, 2020Cancer Biology

Balance of Mechanical Forces Drives Endothelial Gap Formation and May Facilitate Cancer and Immune-Cell Extravasation

J. Escribano, M. Chen, E. Moeendarbary, X. Cao, V. Shenoy, J. M. Garcia-Aznar, R. Kamm, F. Spill

This study shows that mechanical forces drive spontaneous gap formation in endothelial cells, which may facilitate cancer and immune cell extravasation by preferentially occurring at cell vertices.

bioRxiv · Nov 26, 2018Bioengineering