Author

J. Escribano

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Biophysics· 1

Signature Assays, Organisms & Methods

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Approach:mechanistic / dynamical model· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

E. Moeendarbary· 2 shared F. Spill· 2 shared J. M. Garcia-Aznar· 2 shared A. T. Weeraratna· 1 shared E. McEvoy· 1 shared G. E. Marino· 1 shared M. Chen· 1 shared R. D. Kamm· 1 shared

Activity by Year

2018-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersBiophysics

E. Moeendarbary

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Author profile →

2 papers

Recent·Most cited

Modeling the three-way feedback between cellular contractility, actin polymerization, and adhesion turnover resolves the contradictory effects of RhoA and Rac1 on endothelial junction dynamics

E. McEvoy, T. Sneh, E. Moeendarbary, G. E. Marino, X. Chen, J. Escribano, F. Spill, J. M. Garcia-Aznar, A. T. Weeraratna, T. M. Svitkina, R. D. Kamm, V. B. Shenoy

This study develops a chemo-mechanical model to explain how RhoA and Rac1 differentially affect endothelial junction dynamics through their roles in contractility, adhesion turnover, and actin polymerization.

bioRxiv · Mar 15, 2021 · PreprintBiophysics

Balance of Mechanical Forces Drives Endothelial Gap Formation and May Facilitate Cancer and Immune-Cell Extravasation

J. Escribano, M. Chen, E. Moeendarbary, X. Cao, V. Shenoy, J. M. Garcia-Aznar, R. Kamm, F. Spill

This study shows that mechanical forces drive spontaneous gap formation in endothelial cells, which may facilitate cancer and immune cell extravasation by preferentially occurring at cell vertices.

bioRxiv · Nov 26, 2018Bioengineering