Author

Richa Bajpai

St. Jude Children's Research Hospital

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Genetics· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1 Assay:ChIP-seq· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Shondra M. Pruett‐MilleriD· 3 shared Aaron PitreiD· 1 shared Aidan W. BlaniD· 1 shared Andrey TvardovskiyiD· 1 shared Baisakhi MondaliD· 1 shared Beisi XuiD· 1 shared Cheon‐Gil Park· 1 shared David W. BaggettiD· 1 shared

Activity by Year

2021-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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3 shared papersBiochemistry

Shondra M. Pruett‐MillerORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Granular component sub-phases direct ribosome biogenesis in the nucleolus

Priyanka Dogra, Mylene C. Ferrolino, Suparna Khatun, Qi Miao, Michele Tolbert, Aaron Pitre, Shondra M. Pruett‐Miller, Swarnendu Tripathi, David W. Baggett, Jaison John, George E. Campbell, Katelyn Jackson, Richa Bajpai, Toler Freyaldenhoven, Eric B. Gibbs, , Richard W. Kriwacki

The paper uses super-resolution microscopy to show that nucleolus granular component sub-phases are organized into multiphase condensates that facilitate ribosome subunit assembly.

Molecular Cell · Jul 15, 2026Peer-reviewedBiochemistry

Patient derived model of UBA5-associated encephalopathy identifies defects in neurodevelopment and highlights potential therapies

Helen Chen, Yong‐Dong Wang, Aidan W. Blan, Edith P. Almanza Fuerte, Emily S. Bonkowski, Richa Bajpai, Shondra M. Pruett‐Miller, Heather C. Mefford

The paper develops a patient-derived cortical organoid model of UBA5-associated encephalopathy, identifies neurodevelopmental defects, and highlights potential therapeutic strategies.

bioRxiv · Jan 25, 2024 · PreprintGenetics

The MLL3/4 complexes and MiDAC act antagonistically as genome-wide regulators of H4K20ac to control a specific gene expression program

Xiaokang Wang, Wojciech Rosikiewicz, Yurii Sedkov, Baisakhi Mondal, Satish Kallappagoudar, Andrey Tvardovskiy, Richa Bajpai, Beisi Xu, Shondra M. Pruett‐Miller, Robert Schneider, Hans‐Martin Herz

The paper shows that MiDAC and MLL3/4 complexes antagonistically regulate H4K20ac and gene expression through their interaction and co-localization at enhancer regions.

bioRxiv · Dec 17, 2021 · PreprintMolecular Biology