Author

Richard S. Maser

Jackson Laboratory

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioinformatics· 1 Cancer Biology· 1 Genetic & Genomic Medicine· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:gene regulatory network inference· 2 Approach:statistical model· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1

Frequent Co-Authors

Carol J. BultiD· 4 shared Julie M. WellsiD· 3 shared Heidi J. Munger· 2 shared Rosalinda A. Doty· 2 shared Andrés HernándeziD· 1 shared Anne‐Sophie Jourdain· 1 shared Anuj SrivastavaiD· 1 shared Ashley TuckeriD· 1 shared

Activity by Year

2019-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 28 mutual co-author links

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4 shared papersCancer Biology

Carol J. BultORCID

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Author profile →

4 papers

Recent·Most cited

Cell-type Specific Alteration of Dicer1 Accelerates Tumor Progression in Mouse Models of KRAS-driven Lung Adenocarcinoma

Julie M. Wells, Richard S. Maser, Rosalinda A. Doty, Ashley Tucker, Wendy Memishian, Teresa McGee, Naomi Mitchell-Hutchinson, Paige Ramkissoon, Simon Lesbirel, Jeremy R. Charette, Heidi J. Munger, Travis Beckett, Carol J. Bult

This study shows that cell-type-specific Dicer1 disruption accelerates KRAS-driven lung adenocarcinoma progression in mice, highlighting non-cell-autonomous miRNA-mediated effects.

bioRxiv · Jun 1, 2026 · PreprintCancer Biology

Missense variants affecting the actin-binding domains of PLS3 cause X-linked congenital diaphragmatic hernia and body wall defects

Florence Petit, Mauro Longoni, Julie M. Wells, Richard S. Maser, Eric L. Bogenschutz, Matthew J. Dysart, Hannah T. M. Contreras, Frédéric Frénois, Barbara R. Pober, Robin Dawn Clark, Philip F. Giampietro, Hilger H. Ropers, Hao Hu, Marı́a Loscertales, Richard Wagner, , Frances A. High

The paper identifies missense variants in PLS3 causing X-linked congenital diaphragmatic hernia and body wall defects through genetic analysis and functional studies.

The American Journal of Human Genetics · Sep 25, 2023Peer-reviewedGenetic & Genomic Medicine

RNA Immunoprecipitation and Sequencing of Isolated RNAs (RIP-SIR) Identifies Endogenous miRNA-Target Interactions

Julie M. Wells, Anuj Srivastava, Richard S. Maser, Rupesh K. Kesharwani, Vishal Sarsani, Sandra L Daigle, Ryan P. Lynch, Heidi J. Munger, Rosalinda A. Doty, Ruth L. Saxl, Carol J. Bult

The paper introduces RIP-SIR, a method to identify endogenous miRNA-target interactions across tissues and cell types.

bioRxiv · Aug 22, 2022 · PreprintMolecular Biology

Genetic mapping in Diversity Outbred mice identifies a Trpa1 variant influencing late phase formalin response

Jill M. Recla, Jason A. Bubier, Daniel M. Gatti, Jennifer L. Ryan, Katie H. Long, Raymond F. Robledo, Nicole C. Glidden, Guoqiang Hou, Gary A. Churchill, Richard S. Maser, Zhongwei Zhang, Erin E. Young, Elissa J. Chesler, Carol J. Bult

This study identifies a Trpa1 splice junction variant in Diversity Outbred mice that influences late phase formalin response, linking it to pain sensitivity mechanisms.

Pain · Mar 27, 2019Peer-reviewedBioinformatics