Author

S. B. Stephens

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 3

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell signaling & trafficking· 3 Approach:experimental assay· 3 Research Focus:cell type annotation· 1 Assay:electronic health records· 1 Assay:electrophysiology· 1 Organism:human· 1 Assay:imaging· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

C. K. Boyer· 2 shared J. Zhang· 2 shared Y. Wang· 2 shared A. Mishra· 1 shared A. W. Norris· 1 shared C. J. Bauchle· 1 shared C. Kang· 1 shared C.-L. Yu· 1 shared

Activity by Year

2017-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCell Biology

C. K. Boyer

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Synchronized proinsulin trafficking reveals delayed Golgi export accompanies beta-cell secretory dysfunction in a rodent model of hyperglycemia

C. K. Boyer, J. Zhang, Y. Wang, S. B. Stephens

This study uses a rodent model of hyperglycemia to show that proinsulin trafficking is delayed at the Golgi, contributing to beta-cell secretory dysfunction in Type 2 diabetes.

bioRxiv · Oct 31, 2022 · PreprintCell Biology

Nutrient metabolism regulates insulin granule formation in the pancreatic islet β-cell via ER redox homeostasis

K. E. Rohli, C. K. Boyer, S. C. Bearrows, M. R. Moyer, W. S. Elison, C. J. Bauchle, S. E. Blom, C.-L. Yu, M. R. Pope, J. Zhang, Y. Wang, S. B. Stephens

This study shows that nutrient metabolism and ER redox homeostasis regulate insulin granule formation in pancreatic β-cells, linking glucose metabolism to proinsulin export and T2D pathogenesis.

bioRxiv · Sep 22, 2021 · PreprintCell Biology

SWELL1 is a glucose sensor required for β-cell excitability and insulin secretion

C. Kang, S. K. Gunasekar, A. Mishra, L. Xie, Y. Zhang, S. Pai, Y. Gao, A. W. Norris, S. B. Stephens, R. Sah

This study identifies SWELL1 as a glucose sensor in β-cells that links glucose-induced swelling to chloride current activation, which is required for glucose-stimulated insulin secretion.

bioRxiv · Jun 24, 2017Cell Biology