Author

S. Germer

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:cell type annotation· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Assay:fluorescence / live-cell microscopy· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

A. A. Jungbluth· 1 shared A. Beheshti· 1 shared A. C. Borczuk· 1 shared A. Corvelo· 1 shared A. Craney· 1 shared A. D. Jayaprakash· 1 shared A. J. Kriegel· 1 shared A. M. Melnick· 1 shared

Activity by Year

2018-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperCancer Biology

A. A. Jungbluth

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Author profile →

3 papers

Recent·Most cited

A General Method to Accurately Count Molecular Complexes and Determine the Degree of Labelling in Cells Using Protein Tags

S. A. Tashev, J. Euchner, K. Yserentant, S. Hänselmann, F. Hild, W. Chmielewicz, J. Hummert, F. Schwörer, N. Tsopoulidis, S. Germer, Z. Sassmannshausen, O. T. Fackler, U. Klingmüller, D.-P. Herten

This paper introduces ProDOL, a method to measure degree of labelling in cells using protein tags, and applies it to study HIV-1 Nef's effect on CD4 T cell activation.

bioRxiv · Sep 8, 2023 · PreprintCell Biology

Systemic Tissue and Cellular Disruption from SARS-CoV-2 Infection revealed in COVID-19 Autopsies and Spatial Omics Tissue Maps

J. Park, J. Foox, T. Hether, D. Danko, S. Warren, Y. Kim, J. Reeves, D. J. Butler, C. Mozsary, J. Rosiene, A. Shaiber, E. Afshinnekoo, M. MacKay, Y. Bram, V. Chandar, , Schwar

This study identifies systemic tissue and cellular disruption from SARS-CoV-2 infection using autopsy and spatial omics data, showing tissue-specific damage and transcriptional changes.

bioRxiv · Mar 8, 2021 · PreprintMicrobiology

YES1 amplification: a mechanism of acquired resistance to EGFR inhibitors identified by transposon mutagenesis and clinical genomics

P.-D. Fan, G. Narzisi, A. D. Jayaprakash, E. Venturini, N. Robine, P. Smibert, S. Germer, H. A. Yu, E. J. Jordan, P. K. Paik, Y. Y. Janjigian, J. E. Chaft, L. Wang, A. A. Jungbluth, S. Middha, , M. Ladanyi

This study identifies YES1 amplification as a novel mechanism of resistance to EGFR inhibitors in lung cancer using transposon mutagenesis and clinical genomic analysis.

bioRxiv · Mar 17, 2018Cancer Biology