Author

S. Gispert

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Organism:Drosophila· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

A. Maletzko· 1 shared A. P. West· 1 shared B. Lu· 1 shared C. Citron· 1 shared G. Auburger· 1 shared G. W. J. Auburger· 1 shared I. Tantray· 1 shared I. Wittig· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperImmunology

A. Maletzko

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2 papers

Recent·Most cited

Loss of mitochondrial protease CLPP activates type I interferon responses through the mtDNA-cGAS-STING signaling axis

S. Torres-Odio, Y. Lei, S. Gispert, A. Maletzko, J. Key, S. Menissy, I. Wittig, G. W. J. Auburger, A. P. West

This study shows that CLPP deficiency causes mtDNA stress, which activates the cGAS-STING pathway and leads to type I interferon responses and antiviral gene expression.

bioRxiv · Aug 30, 2020Immunology

MISTERMINATE Mechanistically Links Mitochondrial Dysfunction with Proteostasis Failure

Z. Wu, I. Tantray, J. Lim, S. Chen, Y. Li, Z. Davis, C. Citron, J. Dong, S. Gispert, G. Auburger, O. Brandman, X. Bi, M. Snyder, B. Lu

The paper identifies MISTERMINATE as a mechanism linking mitochondrial dysfunction to proteostasis failure through translational termination impairment and C-terminal extension of mitochondrial proteins.

bioRxiv · Feb 19, 2019Cell Biology