Author

S. Hamsanathan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Cell Biology· 1

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Organism:C. elegans· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:metabolism & bioenergetics· 1 Assay:metabolomics· 1 Assay:proteomics· 1

Frequent Co-Authors

A. A. Sims· 1 shared A. U. Gurkar· 1 shared E. Siefken· 1 shared H. Bayir· 1 shared H. L. Pepper· 1 shared H. Shinglot· 1 shared N. W. Snyder· 1 shared P. Sen· 1 shared

Activity by Year

2021-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCell Biology

A. A. Sims

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1 paper

Recent·Most cited

Persistent DNA damage rewires lipid metabolism and promotes histone hyperacetylation via MYS-1/Tip60.

S. Hamsanathan, T. Anthonymuthu, S. J. Mullet, S. Han, H. Shinglot, E. Siefken, A. A. Sims, P. Sen, H. L. Pepper, S. G. Wendell, N. W. Snyder, H. Bayir, V. E. Kagan, A. U. Gurkar

This study shows persistent DNA damage alters lipid metabolism and histone acetylation via MYS-1/Tip60, leading to immune-like responses and age-related decline.

bioRxiv · Jun 24, 2021Cell Biology