Author

H. Bayir

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Assay:proteomics· 2 Organism:C. elegans· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:DNA methylation· 1

Frequent Co-Authors

V. E. Kagan· 2 shared A. A. Sims· 1 shared A. Bhuniya· 1 shared A. Kossenkov· 1 shared A. N. R. Poli· 1 shared A. U. Gurkar· 1 shared B. Bristow· 1 shared C. Hart· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersImmunology

V. E. Kagan

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Targeting LxCxE cleft pocket of retinoblastoma protein in M2 macrophages inhibits ovarian cancer progression

E. N. Tcyganov, T. Kwak, X. Yang, A. N. R. Poli, C. Hart, A. Bhuniya, J. Cassel, A. Kossenkov, N. Auslander, L. Lu, P. Sharma, M. D. G. C. Mendoza, D. Zhigarev, M. G. Cadungog, S. Jean, , L. J. Montaner

This study shows targeting the LxCxE cleft pocket of Rb in M2 macrophages inhibits ovarian cancer progression by reducing immunosuppressive TAMs and enhancing T cell infiltration.

bioRxiv · May 10, 2024 · PreprintImmunology

Persistent DNA damage rewires lipid metabolism and promotes histone hyperacetylation via MYS-1/Tip60.

S. Hamsanathan, T. Anthonymuthu, S. J. Mullet, S. Han, H. Shinglot, E. Siefken, A. A. Sims, P. Sen, H. L. Pepper, S. G. Wendell, N. W. Snyder, H. Bayir, V. E. Kagan, A. U. Gurkar

This study shows persistent DNA damage alters lipid metabolism and histone acetylation via MYS-1/Tip60, leading to immune-like responses and age-related decline.

bioRxiv · Jun 24, 2021Cell Biology