Author

S. Rosario

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 4 Cell Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 4 Approach:statistical model· 4 Research Focus:chromatin & epigenetic regulation· 3 Organism:human· 3 Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Organism:mouse· 2

Frequent Co-Authors

D. Salyakina· 3 shared J. Naipauer· 3 shared S. Williams· 3 shared E. A. Mesri· 2 shared M. Abba· 2 shared A. Rizvi· 1 shared A. Safina· 1 shared C. Premer· 1 shared

Activity by Year

2018-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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3 shared papersCancer Biology

D. Salyakina

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

A non-coding RNA network involved in KSHV tumorigenesis

J. Naipauer, M. E. Garcia Sola, D. Salyakina, S. Rosario, S. Williams, O. Coso, M. C. Abba, E. A. Mesri, E. Lacunza

The paper identifies a non-coding RNA network involving lncRNAs and miRNAs that modulate KSHV tumorigenesis pathways and develops therapeutic target signatures.

bioRxiv · Mar 5, 2021Cancer Biology

RNA-sequencing analysis of a multistep and hit-and-run cell and animal model of KSHV tumorigenesis reveal the roles of mutations, CpG methylation, and viral-infection footprints in oncogenesis.

j. naipauer, S. Rosario, D. Salyakina, G. Journo, S. Williams, M. Abba, M. Shamay, E. A. Mesri

The paper uses RNA-seq and methylation analysis in a KSHV mouse model to show how viral infection and host mutations drive oncogenesis through epigenetic and genetic changes.

bioRxiv · Oct 3, 2019 · PreprintCancer Biology

PDGFRA Defines the Mesenchymal Stem Cell Kaposi’s Sarcoma Progenitors by Enabling KSHV Oncogenesis in an Angiogenic Environment

J. Naipauer, S. Rosario, S. Gupta, C. Premer, O. Mendez, M. Schlesinger, V. Ponzinibbio, V. Jain, L. Gay, R. Renne, H. L. Chan, L. Morey, D. Salyakina, M. Abba, S. Williams, , E. Mesri

The paper identifies PDGFRA+ bone marrow-derived mesenchymal stem cells as Kaposi’s sarcoma progenitors and shows pro-angiogenic conditions enable KSHV oncogenesis through epigenetic de-repression.

bioRxiv · Oct 2, 2019Cancer Biology

Prevention of chromatin destabilization by FACT is crucial for malignant transformation

P. Sandlesh, A. Safina, I. Goswami, L. Prendergast, S. Rosario, E. C. Gomez, J. Wanf, K. V. Gurova

The paper shows that FACT prevents chromatin destabilization in transformed cells, which is crucial for malignant transformation and survival.

bioRxiv · Dec 17, 2018Cell Biology

The miR-96 and RAR&gamma signaling axis governs androgen signaling and prostate cancer progression

M. D. Long, P. K. Singh, J. R. Russell, G. Llimos, S. Rosario, A. Rizvi, H. Withers, P. R. van den Berg, J. Kirk, L. E. Sucheston-Campbell, D. J. Smiraglia, M. J. Campbell

This study identifies miR-96 as a regulator of RARγ expression, which in turn controls androgen signaling and prostate cancer progression through gene network alterations.

bioRxiv · Apr 26, 2018Cancer Biology