Author

S. Zanotti

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Approach:experimental assay· 1 Organism:mouse· 1 Assay:multiome· 1 Assay:proteomics· 1

Frequent Co-Authors

B. Decaesteker· 1 shared C. Bragato· 1 shared C. Introna· 1 shared C. Van Neste· 1 shared D. Deforce· 1 shared F. Blasevich· 1 shared F. Speleman· 1 shared F. Van Nieuwerburgh· 1 shared

Activity by Year

2018-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCancer Biology

B. Decaesteker

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Author profile →

2 papers

Recent·Most cited

MYCN-induced nucleolar stress drives an early senescence-like transcriptional program in hTERT-immortalized RPE cells

S. Zanotti, S. Vanhauwaert, C. Van Neste, V. Olexiouk, J. Van Laere, M. Verschuuren, L. M. Mus, K. Durinck, L. Tilleman, D. Deforce, F. Van Nieuwerburgh, M. D. Hogarty, B. Decaesteker, W. H. De Vos, F. Speleman

This study shows that MYCN activation in RPE cells causes nucleolar stress and a senescence-like transcriptional program without full senescence hallmarks, leading to growth attenuation.

bioRxiv · Jan 22, 2021Cancer Biology

Exosomes and exosomal miRNAs from muscle-derived fibroblasts promote skeletal muscle fibrosis

M. Mora, S. Zanotti, S. Gibertini, F. Blasevich, C. Bragato, S. Saredi, C. Introna, P. Bernasconi, L. Maggi, R. Mantegazza

This study shows that exosomes from DMD fibroblasts promote fibrosis by transferring miR-199a-5p, which reduces caveolin-1 and activates fibroblast-to-myofibroblast conversion.

bioRxiv · Feb 19, 2018Pathology