Author

Sadaf Fazeli

Karolinska Institutet

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:protein structure prediction· 2 Assay:proteomics· 2 Research Focus:benchmarking· 1 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Ann-Charlotte BjörklundiD· 2 shared Christina Maria Perraki· 2 shared Indira BarbosaiD· 2 shared Janne LehtiöiD· 2 shared Mikaela Hillberg Widfeldt· 2 shared Pedro Luís MouraiD· 2 shared Rui Mamede BrancaiD· 2 shared Teresa Mortera BlancoiD· 2 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersPhysiology

Ann-Charlotte BjörklundORCID

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2 papers

Recent·Most cited

Proteomics of steady-state and culture-derived human erythropoiesis reveals the dynamics of erythroid stress

Christina Maria Perraki, Kovi Rishya Shrung, Sadaf Fazeli, Yaroslav Kaminskiy, Ann-Charlotte Björklund, Indira Barbosa, Mikaela Hillberg Widfeldt, Janne Lehtiö, Eva Hellström‐Lindberg, Teresa Mortera Blanco, Rui Mamede Branca, Pedro Luís Moura

This study compares proteomic profiles of steady-state and cultured human erythroid progenitors, revealing how culture-induced stress alters protein expression and erythroid maturation dynamics.

bioRxiv · Oct 14, 2026 · PreprintPhysiology

RNA splicing factor mutations drive myeloid neoplasm oncogenesis through protein complex poisoning

Pedro Luís Moura, Rui Mamede Branca, Yaroslav Kaminskiy, Ioannis Siavelis, Kovi R Shrung, Sophia Hofmann, Christina Maria Perraki, Alexandra Argyriou, Sadaf Fazeli, Masahiro M Nakagawa, Teresa Mortera Blanco, Steffen Boettcher, Caroline Kubaczka, Thorsten M. Schlaeger, Maria Creignou, , Eva Hellström-Lindberg

This study identifies how RNA splicing factor mutations drive myeloid neoplasm by disrupting proteomic networks through mis-splicing, validated via single-cell transcriptomics and proteomics.

bioRxiv · Sep 30, 2026 · PreprintCancer Biology