Author

Stephen D. Dertinger

Litron Laboratories (United States)

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Assay:DNA methylation· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1

Frequent Co-Authors

Dorothea K. Torous· 3 shared Bevin P. EngelwardiD· 2 shared Ilana S. NazariiD· 2 shared Jennifer E. KayiD· 2 shared John M. EssigmanniD· 2 shared Joshua J. CorriganiD· 2 shared Leona D. SamsoniD· 2 shared Lindsay B. VolkiD· 2 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersCancer Biology

Dorothea K. Torous

Mutual co-authors in cluster

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3 papers

Recent·Most cited

INVESTIGATION OF ETHYLENE OXIDE GENOTOXICITY DOSE-RESPONSE TO INFORM CANCER RISK ASSESSMENT

B. Bhaskar Gollapudi, James S. Bus, Phillip Cassidy, Jeffrey T. Weinberg, Jeffrey C. Bemis, Dorothea K. Torous, Stephen D. Dertinger, Kun Lü, Richard Reiss, Abby A. Li

This study investigates the dose-response relationship of ethylene oxide-induced genetic damage in mice to assess its potential carcinogenicity through a mutagenic mechanism.

Environmental and Molecular Mutagenesis · Jun 1, 2026Peer-reviewedPharmacology & Toxicology

Persistent interferon signaling and clonal expansion mark early events in DNA methylation damage-induced liver cancer

Lee J. Pribyl, Jennifer E. Kay, Joshua J. Corrigan, Lindsay B. Volk, Monét Norales, Norah Auma Owiti, Evan A Kowal, Ishwar N. Kohale, Ilana S. Nazari, Mallory Swanson, Aimee C. Moise, Duanduan Ma, Stuart S. Levine, Emily M Michelsen, Robert G. Croy, , Bevin P. Engelward

This study identifies persistent interferon signaling and clonal expansion as early events in NDMA-induced liver cancer by analyzing molecular changes in mice.

NAR Cancer · Apr 8, 2026Peer-reviewedCancer Biology

Sex and Alkyladenine DNA Glycosylase Expression are Key Susceptibility Factors for NDMA-induced Mutations, Toxicity, and Cancer

Jennifer E. Kay, Joshua J. Corrigan, Lindsay B. Volk, Amanda L. Armijo, Ilana S. Nazari, Dorothea K. Torous, Svetlana L. Avlasevich, Robert G. Croy, Dushan N. Wadduwage, Stephen D. Dertinger, John M. Essigmann, Leona D. Samson, Bevin P. Engelward

This study investigates sex differences in susceptibility to NDMA-induced mutations, toxicity, and cancer in mice, finding males are more prone to cancer while Aag overexpression increases toxicity, particularly in females.

bioRxiv · May 13, 2025 · PreprintCancer Biology