Author

T. Chattopadhyay

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:metabolism & bioenergetics· 2 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

A. Bhansali· 1 shared A. Chakraborty· 1 shared A. Choubey· 1 shared A. K. Kar· 1 shared B. Maniyadath· 1 shared B. Medhi· 1 shared C. K. Mantri· 1 shared D. Ghosh· 1 shared

Activity by Year

2019-2019 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperPhysiology

A. Bhansali

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2 papers

Recent·Most cited

Hyperinsulinemia promotes HMGB1 release leading to inflammation induced systemic insulin resistance: An interplay between pancreatic beta-cell and peripheral organs

A. Choubey, A. K. Kar, K. Girdhar, T. Chattopadhyay, S. Dogra, S. Kushwaha, B. Medhi, A. Bhansali, C. K. Mantri, U. K. Seetharam, D. Ghosh, P. Mondal

This study shows hyperinsulinemia promotes inflammation via HMGB1 and TLR4 signaling, leading to systemic insulin resistance, and demonstrates naltrexone's protective effects.

bioRxiv · Jul 17, 2019 · PreprintPhysiology

Temporal gating of SIRT1 functions by O-GlcNAcylation prevents hyperglycemia and enables physiological transitions in liver

T. Chattopadhyay, B. Maniyadath, H. P. Bagul, A. Chakraborty, N. Shukla, S. Budnar, U. Kolthur-Seetharam

This study identifies nutrient-dependent O-GlcNAcylation of SIRT1 as a mechanism controlling hepatic metabolism and insulin signaling during fasted-to-refed transitions.

bioRxiv · Apr 2, 2019 · PreprintPhysiology