Author

T. Schild

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 2 Immunology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:metabolism & bioenergetics· 2 Assay:metabolomics· 2 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1 Organism:mouse· 1

Frequent Co-Authors

A. P. Gomes· 2 shared D. Ilter· 2 shared V. Low· 2 shared A. Ahmed· 1 shared A. Baako· 1 shared A. Lipschutz· 1 shared A. Mutvei· 1 shared A. Rosenzweig· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

A. P. Gomes

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Early life high fructose exposure disrupts microglia function and impedes neurodevelopment

Z. Wang, A. Lipschutz, Z.-L. Liu, A. Trzeciak, I. Miranda, C. Martinez de la Torre, T. Schild, T. Lazarov, W. S. Rojas, P. Saavedra, J. Romero-Pichardo, A. Baako, F. Geissmann, G. Faraco, L. Gan, , J. Perry

This study shows that early life high fructose exposure impairs microglia function and neurodevelopment, with GLUT5 deletion reversing these effects.

bioRxiv · Aug 14, 2023 · PreprintImmunology

NADK upregulation is an essential metabolic adaptation that enables breast cancer metastatic colonization

D. Ilter, T. Schild, N. Ward, S. Drapela, E. Adhikari, V. Low, E. Le-Lau, G. M. DeNicola, M. McReynolds, A. P. Gomes

This study identifies NADK upregulation as a critical metabolic adaptation enabling breast cancer metastasis by enhancing NADPH production and oxidative stress resistance.

bioRxiv · Mar 25, 2022 · PreprintCancer Biology

Age-induced methylmalonic acid accumulation promotes tumor progression and aggressiveness

A. P. Gomes, D. Ilter, V. Low, J. Endress, J. Fernandez-Garcia, A. Rosenzweig, T. Schild, D. Broekaert, A. Ahmed, M. Planque, I. Elia, J. Han, C. Kinzig, E. Mullarky, A. Mutvei, , J. Blenis

The paper shows that methylmalonic acid accumulation with age promotes tumor progression by inducing SOX4 and reprogramming cancer cells.

bioRxiv · Jun 4, 2020 · PreprintCancer Biology