Author

T. Takenaga

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Assay:electronic health records· 1 Organism:human· 1 Assay:organoid / primary culture· 1

Frequent Co-Authors

T. Noda· 2 shared Y. Muramoto· 2 shared A. Hirbayashi· 1 shared C. Onishi· 1 shared K. Fujimoto· 1 shared K. Toyooka· 1 shared K. Yamauchi· 1 shared M. Nakano· 1 shared

Activity by Year

2023-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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2 shared papersMicrobiology

T. Noda

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Coatomer complex I is required for the transport of SARS-CoV-2 progeny virions from the endoplasmic reticulum-Golgi intermediate compartment

A. Hirbayashi, Y. Muramoto, T. Takenaga, Y. Tsunoda, M. Wakazaki, M. Sato, Y. Fujita-Fujiharu, N. Nomura, K. Yamauchi, C. Onishi, M. Nakano, K. Toyooka, T. Noda

This study identifies COPI as essential for SARS-CoV-2 virion transport from the ERGIC, showing that COPB2 depletion inhibits viral replication.

mBio · Jan 8, 2025Peer-reviewedMicrobiology

Isogenic iPSC-derived proximal and distal lung-on-chip models: Tissue- and virus-specific immune responses in human lungs

S. Yadav, K. Fujimoto, T. Takenaga, S. Takahashi, Y. Muramoto, R. Mikawa, T. Noda, S. Gotoh, R. Yokokawa

The paper develops isogenic lung-on-chip models from iPSC-derived cells to study tissue-specific immune responses to SARS-CoV-2 and Influenza viruses.

bioRxiv · Nov 24, 2023 · PreprintBioengineering