Author

W. Pu

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:mouse· 3 Research Focus:cell type annotation· 2 Research Focus:cell-cell communication· 2 Research Focus:differential expression· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:clustering· 1

Frequent Co-Authors

A. Gillich· 1 shared A. Ng· 1 shared A. Vaughan· 1 shared B. Jardin· 1 shared B. Zhou· 1 shared C. Guo· 1 shared C. Wu· 1 shared C. Zhao· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 10 mutual co-author links

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1 shared paperCell Biology

A. Gillich

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Author profile →

4 papers

Recent·Most cited

Non-cell autonomous cardiomyocyte regulation complicates gene supplementation therapy for LMNA cardiomyopathy

Y. Sun, C. Guo, Z. Chen, J. Lin, L. Yang, Y. Zhang, C. Wu, D. Zhao, B. Jardin, W. Pu, M. Zhao, E. Dong, X. Hu, S. Zhang, Y. Guo

This study shows that gene supplementation for LMNA cardiomyopathy is ineffective when targeting cardiomyocytes alone, as non-cell autonomous effects dominate, suggesting alternative therapeutic targets.

bioRxiv · Jul 18, 2023 · PreprintCell Biology

Airway secretory cell-derived p63+ progenitors contribute to alveolar regeneration after sterile lung injury

Z. Lv, Z. Liu, K. Liu, W. Pu, Y. Li, H. Zhao, Y. Xi, A. Vaughan, A. Gillich, B. Zhou

bioRxiv · Feb 27, 2023 · PreprintCell Biology

Inhibition of HSP90 reversed STAT3 mediated muscle wasting induced by cancer cachexia

H. Wang, M. Niu, S. Song, Z. Su, L. Wei, L. Li, W. Pu, C. Zhao, Y. Ding, W. Cao, Q. Gao

The paper shows that inhibiting HSP90 reduces STAT3-mediated muscle wasting in cancer cachexia by disrupting the HSP90/STAT3/FOXO1 axis.

bioRxiv · Jan 27, 2021 · PreprintPathology

Robust differentiation of human pluripotent stem cells into endothelial cells via temporal modulation of ETV2 with modified mRNA

K. Wang, R.-Z. Lin, X. Hong, A. Ng, C.-N. Lee, J. Neumeyer, G. Wang, X. Wang, M. Ma, W. Pu, G. Church, J. Melero-Martin

This study develops a method to efficiently generate human-induced pluripotent stem cell-derived endothelial cells by modulating ETV2 with modified mRNA, achieving high consistency and functionality.

bioRxiv · Mar 2, 2020Cell Biology