Author

Y. Chin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 3

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cancer & tumor microenvironment· 3 Approach:experimental assay· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

R. Benezra· 2 shared A. Gower· 1 shared A. H. Skalet· 1 shared A. Iavarone· 1 shared A. J. Dannenberg· 1 shared A. Lasorella· 1 shared A. Sapre· 1 shared A. Sattler· 1 shared

Activity by Year

2017-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

R. Benezra

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Relevance of Circulating Hybrid Cells as a Non-Invasive Biomarker for Myriad Solid Tumors

M. S. Dietz, T. L. Sutton, B. S. Walker, C. E. Gast, L. Zarour, S. K. Sengupta, J. R. Swain, J. Eng, M. Parappilly, K. Limbach, A. Sattler, E. Burlingame, Y. Chin, A. Gower, J. L. Montoya Mira, , M. H. Wong

This study identifies circulating hybrid cells (CHCs) as a novel biomarker for metastatic solid tumors, showing they outnumber CTCs and exhibit tumor-initiating properties across multiple cancer types.

bioRxiv · Mar 11, 2021 · PreprintCancer Biology

Anti-tumor effects of an Id antagonist with no acquired resistance

P. M. Wojnarowicz, M. G. Escalono, Y.-H. Huang, B. Desai, Y. Chin, R. Shah, S. Xu, O. Ouerfelli, R. K. Soni, J. Philip, D. C. Montrose, J. H. Healey, V. K. Rajasekhar, W. A. Garland, L. Norton, , R. Benezra

This study demonstrates that AGX51, an Id antagonist, effectively inhibits tumor growth in multiple cancer models without inducing acquired resistance.

bioRxiv · Jan 6, 2020 · PreprintCancer Biology

Whole chromosome loss in tetraploid cells confers tumorigenic potential in a mouse allograft model

R. Thomas, D. H. Marks, Y. Chin, R. Benezra

This study shows that whole chromosome loss in tetraploid cells leads to tumorigenic potential in a mouse model by increasing chromosomal instability and DNA damage.

bioRxiv · Aug 4, 2017Cancer Biology