Author

O. Ouerfelli

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

N. Rosen· 2 shared A. Iavarone· 1 shared A. J. Dannenberg· 1 shared A. Lasorella· 1 shared B. Desai· 1 shared C. Amor· 1 shared D. C. Montrose· 1 shared D. Warner· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersCancer Biology

N. Rosen

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2 papers

Recent·Most cited

eIF4A controls translation of estrogen receptor alpha and is a therapeutic target in advanced breast cancer

J. A. Boyer, M. Sharma, M. Dorso, N. Mai, C. Amor, J. M. Reiter, R. Kannan, S. Gadal, J. Xu, M. Miele, L. Zhuoning, X. Chen, Q. Chang, F. Pareja, S. Worland, , N. Rosen

This paper identifies eIF4A as a key regulator of ER translation in breast cancer and shows that inhibiting eIF4A, either alone or with fulvestrant, suppresses ER expression and tumor growth in advanced ER+ breast cancer.

bioRxiv · May 8, 2024Cancer Biology

Anti-tumor effects of an Id antagonist with no acquired resistance

P. M. Wojnarowicz, M. G. Escalono, Y.-H. Huang, B. Desai, Y. Chin, R. Shah, S. Xu, O. Ouerfelli, R. K. Soni, J. Philip, D. C. Montrose, J. H. Healey, V. K. Rajasekhar, W. A. Garland, L. Norton, , R. Benezra

This study demonstrates that AGX51, an Id antagonist, effectively inhibits tumor growth in multiple cancer models without inducing acquired resistance.

bioRxiv · Jan 6, 2020 · PreprintCancer Biology