Author

D. C. Montrose

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Assay:DNA methylation· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Iavarone· 1 shared A. J. Dannenberg· 1 shared A. Lasorella· 1 shared B. Desai· 1 shared J. H. Healey· 1 shared J. Li· 1 shared J. Massague· 1 shared J. Philip· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperCancer Biology

A. Iavarone

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2 papers

Recent·Most cited

p53 engages the cGAS/STING cytosolic DNA sensing pathway for tumor suppression

M. Ghosh, S. Saha, J. Li, D. C. Montrose, L. A. Martinez

This study shows p53 suppresses tumors by promoting TREX1 degradation, leading to cytosolic DNA accumulation and activation of the cGAS/STING pathway, which induces type I interferons.

bioRxiv · May 26, 2022Cancer Biology

Anti-tumor effects of an Id antagonist with no acquired resistance

P. M. Wojnarowicz, M. G. Escalono, Y.-H. Huang, B. Desai, Y. Chin, R. Shah, S. Xu, O. Ouerfelli, R. K. Soni, J. Philip, D. C. Montrose, J. H. Healey, V. K. Rajasekhar, W. A. Garland, L. Norton, , R. Benezra

This study demonstrates that AGX51, an Id antagonist, effectively inhibits tumor growth in multiple cancer models without inducing acquired resistance.

bioRxiv · Jan 6, 2020 · PreprintCancer Biology