Author

Y. Zhi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:metabolism & bioenergetics· 1 Assay:proteomics· 1

Frequent Co-Authors

X. Li· 2 shared Y. Zheng· 2 shared B. Niu· 1 shared C. D. Dong· 1 shared F. Ma· 1 shared G. Jin· 1 shared H. Liu· 1 shared K. Liu· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersCell Biology

X. Li

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2 papers

Recent·Most cited

Protein S-glutathionylation confers cell resistance to ferroptosis

Y. Ju, Y. Zhang, Y. Qiao, X. Tian, Y. Zheng, T. Yang, B. Niu, X. Li, L. Yu, Z. Liu, Y. Wu, Y. Zhi, Y. Dong, Q. Xu, X. Wang, , X. Li

This study shows that protein S-glutathionylation protects cells from ferroptosis by maintaining ARF6 function and reducing labile iron pools.

bioRxiv · May 3, 2024 · PreprintCell Biology

Molecular mechanism of SARS-Cov-2 components caused ARDS in murine model

T. Gu, S. Zhao, G. Jin, M. Song, Y. Zhi, R. Zhao, F. Ma, Y. Zheng, K. Wang, H. Liu, M. Xin, W. Han, X. Li, C. D. Dong, K. Liu, Z. Dong

This study develops a non-infectious murine model to study SARS-CoV-2-induced ARDS and CSS, and identifies potential therapeutic agents that alleviate inflammatory symptoms.

bioRxiv · Jun 7, 2020 · PreprintPathology