Anne M. Villeneuve

The paper investigates how β-tubulin isotypes TBB-1 and TBB-2 contribute to acentrosomal spindle bipolarity and function in C. elegans oocytes.

The paper reports extreme programmed DNA elimination in Auanema rhodense during embryogenesis, revealing extensive loss of germline-specific DNA from somatic cells.

This study identifies COSA-2 as essential for maintaining crossover-specific repair compartments during meiotic crossover maturation in C. elegans.

The paper investigates the timing and mechanism of crossover designation in C. elegans using advanced imaging techniques, revealing a multi-step, dynamic regulation process.

This study shows that DUO-1, a deubiquitinase in C. elegans, is essential for maintaining meiotic chromosome structures throughout prophase, ensuring proper meiosis.

This study investigates the distinct roles of RAD-54.L and RAD-54.B in C. elegans meiotic prophase, showing RAD-54.L facilitates RAD-51 removal while RAD-54.B prevents excessive RAD-51 accumulation.

The paper identifies DSB-3 as a meiotic DSB-promoting protein in C. elegans, showing both conservation with and divergence from homologous complexes in other organisms.

This study identifies the HAL-2/HAL-3 complex as essential for regulating Polo-like kinase activity during C. elegans meiosis, ensuring proper homolog pairing and synaptonemal complex assembly.

This study identifies NBS-1 as essential for processing meiotic DSBs to promote crossover formation and antagonize error-prone repair, ensuring genome integrity.

The paper analyzes early meiotic prophase events in C. elegans to understand homolog pairing and synapsis mechanisms.

This study shows that meiotic recombination modulates the stability of the synaptonemal complex in C. elegans, with PLK-2 playing a key role in this process.