Author

Yohei Okada

Aichi Medical UniversityORCID 0000-0001-5253-8556Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:protein structure prediction· 1 Assay:proteomics· 1

Frequent Co-Authors

Akinobu OtaiD· 2 shared Hideyuki OkanoiD· 2 shared Kazunari OnoderaiD· 2 shared Yoshitaka HosokawaiD· 2 shared Atsushi ToyodaiD· 1 shared Daisuke Shimojo· 1 shared Gen SobueiD· 1 shared Haruhisa InoueiD· 1 shared

Activity by Year

2020-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersNeuroscience

Akinobu OtaORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

NeurosciencebioRxivPreprintDec 13, 2025

Early pathogenesis of spinal and bulbar muscular atrophy uncovered by human iPSC-derived motor neurons highlights pathogenic neuropeptides as therapeutic targets

Kazunari Onodera, Yuichi Riku, Daisuke Shimojo…·Yohei Okada

The paper uses human iPSC-derived motor neurons to study SBMA, identifying neuropeptides as therapeutic targets.

DOI 10.64898/2025.12.10.692869

humanbulk RNA-seq

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NeurosciencebioRxivPreprintMar 24, 2021

C9ORF72 dipeptide repeat proteins disrupt formation of GEM bodies and induce aberrant accumulation of survival of motor neuron protein

Yuma Kato, Minnie Yokogawa, Ikuma Nakagawa…·Hitomi Tsuiji

This study shows that dipeptide repeat proteins from C9ORF72 mutations disrupt GEM body formation and cause SMN protein mislocalization in motor neurons.

3 citations

humanproteomics

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Molecular BiologyCell ReportsPeer-reviewedJan 1, 2020

Tandem paired nicking promotes precise genome editing with scarce interference by p53

Toshinori Hyodo, Md Lutfur Rahman, Sivasundaram Karnan…·Hiroyuki Konishi

This study introduces tandem paired nicking, a genome editing method that minimizes indels and p53 activation while maintaining knock-in efficiency.

40 citations

experimental assay

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