Author

Kazunari Onodera

Aichi Medical UniversityORCID 0000-0002-3274-0260Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:protein structure prediction· 1 Assay:proteomics· 1

Frequent Co-Authors

Hideyuki OkanoiD· 2 shared Yohei OkadaiD· 2 shared Akinobu OtaiD· 1 shared Daisuke Shimojo· 1 shared Gen SobueiD· 1 shared Haruhisa InoueiD· 1 shared Hitomi TsuijiiD· 1 shared Ikuma Nakagawa· 1 shared

Activity by Year

2021-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersNeuroscience

Hideyuki OkanoORCID

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2 papers

Recent·Most cited

NeurosciencebioRxivPreprintDec 13, 2025

Early pathogenesis of spinal and bulbar muscular atrophy uncovered by human iPSC-derived motor neurons highlights pathogenic neuropeptides as therapeutic targets

Kazunari Onodera, Yuichi Riku, Daisuke Shimojo…·Yohei Okada

The paper uses human iPSC-derived motor neurons to study SBMA, identifying neuropeptides as therapeutic targets.

DOI 10.64898/2025.12.10.692869

humanbulk RNA-seq

𝕏 PostBluesky

NeurosciencebioRxivPreprintMar 24, 2021

C9ORF72 dipeptide repeat proteins disrupt formation of GEM bodies and induce aberrant accumulation of survival of motor neuron protein

Yuma Kato, Minnie Yokogawa, Ikuma Nakagawa…·Hitomi Tsuiji

This study shows that dipeptide repeat proteins from C9ORF72 mutations disrupt GEM body formation and cause SMN protein mislocalization in motor neurons.

3 citations

humanproteomics

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