Richard J. Bennett

This study identifies an IL-17-DUOX2 axis that regulates C. albicans colonization and filamentation in the gut through interactions with candidalysin toxin.

This study identifies Mnl1 as a repressor of azole tolerance in Candida albicans by linking it to sphingolipid metabolism, mitochondrial activity, and PKA signaling.

This study uses RNA sequencing on 21 Candida albicans isolates to link transcriptional variation to phenotypic traits, identifying gene modules associated with virulence and uncovering novel regulators of growth and filamentation.

This study analyzes the genome of Candida albicans to show that mutation and cryptic sexual recombination drive its evolution, with non-random distribution of SNPs and LOH events.

This study analyzes mutations in Candida albicans during microevolution, finding that de novo substitutions and LOH events drive short-term evolution, while purifying selection is strong during host infection.