Author

Hao Wu

University of PennsylvaniaORCID 0000-0001-7804-8205Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Hao Wu24 papers · University of Pennsylvania

Biology Disciplines

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Genetics· 2 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Assay:ChIP-seq· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1 Assay:spatial transcriptomics· 1

Frequent Co-Authors

Emily R. FeiermaniD· 2 shared Erica KorbiD· 2 shared Kyuhyun ChoiiD· 2 shared Marc Vincent FuccilloiD· 2 shared Qi QiuiD· 2 shared Alekh ParanjapyeiD· 1 shared Anna J. VossiD· 1 shared Annabel K. SangreeiD· 1 shared

Activity by Year

2018-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 32 mutual co-author links

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2 shared papersGenetics

Emily R. FeiermanORCID

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3 papers

Recent·Most cited

Histone variant H2BE controls activity-dependent gene expression and homeostatic scaling

Emily R. Feierman, Alekh Paranjapye, Annabel K. Sangree, Rili Ahmad, Marissa J. Maroni, Qi Qiu, Kyuhyun Choi, Marc Vincent Fuccillo, Hao Wu, Erica Korb

This study shows that H2BE is required for activity-dependent gene expression and homeostatic scaling in neurons.

Cell Reports · Dec 19, 2025Peer-reviewedGenetics

Histone variant H2BE enhances chromatin accessibility in neurons to promote synaptic gene expression and long-term memory

Emily R. Feierman, Sean Louzon, Nicholas A. Prescott, Tracy Biaco, Qingzeng Gao, Qi Qiu, Kyuhyun Choi, Katherine C. Palozola, Anna J. Voss, Shreya D. Mehta, Camille N. Quaye, Katherine T. Lynch, Marc Vincent Fuccillo, Hao Wu, Yael David, Erica Korb

This study identifies H2BE as a widely expressed H2B variant that enhances chromatin accessibility in neurons, promoting synaptic gene expression and long-term memory.

Molecular Cell · Jul 17, 2024Peer-reviewedGenetics

Loss of SDHB reprograms energy metabolisms and inhibits high fat diet induced metabolic syndromes

Chenglong Mu, Biao Ma, Chuanmei Zhang, Guangfeng Geng, Xinling Zhang, Linbo Chen, Meng Wang, Jie Li, Tian Zhao, Hongcheng Cheng, Qianping Zhang, Kaili Ma, Qian Luo, Rui Chang, Qiangqiang Liu, , Yushan Zhu

This study shows that SDHB deficiency reprograms energy metabolism and protects against high fat diet-induced obesity by restoring glucose and lipid metabolism.

bioRxiv · Feb 3, 2018 · PreprintMolecular Biology