Author

Victoria Miller-Browne

Harvard UniversityORCID 0000-0001-7845-1985Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Biophysics· 1 Genetic & Genomic Medicine· 1 Genetics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Albert L. MiskoiD· 2 shared Madison SangsteriD· 2 shared Yulia GrishchukiD· 2 shared Brendan R. TobiniD· 1 shared Bryce J. Katch· 1 shared Dan ZhangiD· 1 shared Emily C. BeckwittiD· 1 shared Gabriella N. L. ChuaiD· 1 shared

Activity by Year

2021-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersGenetic & Genomic Medicine

Albert L. MiskoORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

BiophysicsCellPeer-reviewedJan 28, 2026

A non-catalytic role for RFC in PCNA-mediated processive DNA synthesis

Gabriella N. L. Chua, Emily C. Beckwitt, Victoria Miller-Browne…·Shixin Liu

This study reveals a non-catalytic role for RFC in stabilizing PCNA-Polδ interactions during DNA synthesis, essential for processive replication.

3 citationstop 4%

experimental assaychromatin & epigenetic regulation

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Genetic & Genomic MedicinemedRxivPreprintJul 29, 2024

Plasma Proteomic Signature of Mucolipidosis Type IV

Brendan R. Tobin, Albert L. Misko, Victoria Miller-Browne…·Levi Benjamin Wood

The paper identifies plasma proteomic signatures associated with MLIV, showing decreased neuronal proteins and increased muscle proteins, with specific markers correlating with disease severity.

DOI 10.1101/2024.07.29.24311030

humanproteomics

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GeneticsHuman Molecular GeneticsPeer-reviewedApr 2, 2021

MCOLN1 gene-replacement therapy corrects neurologic dysfunction in the mouse model of mucolipidosis IV.

Samantha DeRosa, Monica Salani, Sierra Smith…·Yulia Grishchuk

This study demonstrates that AAV-mediated MCOLN1 gene transfer improves motor function and reduces brain pathology in a mouse model of mucolipidosis IV.

20 citations

mouseexperimental assay

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