Michael J. Whalen

The paper shows that PV-INs have unique metabolic specializations lost after TBI, which can be rescued by 2-deoxyglucose, and identifies PGC-1α and lncRNAs as key factors in this dysfunction.

This study shows that traumatic brain injury disrupts functional connectivity in mice, with recovery and state-dependent changes in connectivity and theta power during stillness.

This study investigates mitochondrial dysregulation's role in neurodegeneration after contusion injury using a 3D in vitro triculture model of human brain cells.

This study shows that subarachnoid hemorrhage in mice causes early and lasting changes in functional connectivity and cognitive performance.

In vivo treatment with 2-deoxyglucose after traumatic brain injury in mice prevented cortical hyperexcitability, synaptic changes, and loss of GABAergic interneurons.