NeurosciencebioRxivPreprint
The A53T mutation in α-synuclein enhances pro-inflammatory activation in human microglia
Marine Krzisch, Bingbing Yuan, Wenyu Chen…·Rudolf Jaenisch
This study shows that the A53T mutation in α-synuclein increases pro-inflammatory activation and oxidative stress in human microglia, potentially contributing to early-onset Parkinson's disease.
2 citations