Nathalie Pujol

The paper shows DPY-6 guides periodic furrow formation in C. elegans cuticle by acting as a molecular mold during molts.

This study shows that C. elegans distinguishes pathogen proximity through compound types, activating EV-dependent physiological responses tailored to immediate or anticipated threats.

The paper describes a novel membrane contact site in mouse type I vestibular hair cells associated with atypical plasma membrane-apposed mitochondria, potentially modulating non-quantal transmission.

The paper identifies spia-1 as a gene involved in immune activation in C. elegans by sensing cuticle damage through its secreted protein with a novel cysteine-cradle domain.

This study identifies the localization and function of C. elegans epicuticlins in the apical extracellular matrix, showing their role in wound repair.

YAAT identifies functionally-related gene groups by integrating co-expression and phylogenetic conservation data, revealing biases in gene set enrichment interpretation.

The paper evaluates tissue-specific RNAi strains in C. elegans and identifies a suitable genetic background for constructing robust tissue-specific RNAi strains.

This study shows that ins-11, an insulin peptide gene in C. elegans, is upregulated by different pathogens in specific tissues and influences immune responses and longevity.