Author

Alessia Ruggieri

Heidelberg UniversityORCID 0000-0001-9981-3308Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:differential expression· 2 Research Focus:host-pathogen & immune response· 2 Assay:proteomics· 2 Approach:wet-lab / biochemical assay· 2 Assay:bulk RNA-seq· 1

Frequent Co-Authors

Nicolas LockeriD· 2 shared Amina Yasmin· 1 shared Belinda Hall· 1 shared Chia Ching WuiD· 1 shared Christoph DieterichiD· 1 shared Doris LindneriD· 1 shared Fotini Nina PapavasiliouiD· 1 shared Francesca TuortoiD· 1 shared

Activity by Year

2018-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersMicrobiology

Nicolas LockerORCID

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3 papers

Recent·Most cited

BiochemistryNucleic Acids ResearchPeer-reviewedJan 7, 2026

The two-step purification method ViREn identifies a single NSUN6-mediated 5-methylcytosine modification promoting dengue virus RNA genome turnover

Chia Ching Wu, Isabel S. Naarmann‐de Vries, Jonas Hartmann…·Alessia Ruggieri

ViREn is a two-step method that purifies dengue virus RNA with high purity, revealing a 5-methylcytosine modification mediated by NSUN6 that affects RNA turnover.

2 citations

bulk RNA-seqexperimental assay

𝕏 PostBluesky

MicrobiologybioRxivPreprintOct 30, 2025

The assembly of stress granule-like foci during foot-and-mouth disease virus infection is uncoupled from activation of cellular intrinsic antiviral signalling

Mariana Marques, Zhaozhi Sun, Amina Yasmin…·Nicolas Locker

This study shows that stress granule formation during FMDV infection is independent of innate immune activation, and that preventing their disassembly reduces viral replication.

DOI 10.1101/2025.10.30.685519

proteomicsexperimental assay

𝕏 PostBluesky

MicrobiologybioRxivPreprintDec 10, 2018

Norovirus infection results in assembly of virus-specific G3BP1 granules and evasion of eIF2α signaling.

Michèle Brocard, Valentina Iadevaia, Philipp Klein…·Nicolas Locker

This study shows that norovirus infection leads to the formation of virus-specific granules and disrupts eIF2α signaling to evade host stress responses.

DOI 10.1101/490318

mouseproteomics

𝕏 PostBluesky