Sangjin Yoo

This study shows that partial FAM19A5 deficiency in mice causes spine maturation issues, hyperactivity, and altered fear responses.

This study examines FAM19A5 expression and function in mice, finding low peripheral levels and no significant interaction with S1PR2.

This study shows that reducing FAM19A5 in Alzheimer's mouse models lowers Aβ plaques and improves cognition, suggesting anti-FAM19A5 antibodies as a potential therapy.

This paper shows that targeting FAM19A5 with a monoclonal antibody improves synaptic function and cognition in Alzheimer's disease models.

This study develops a mouse model to study focused ultrasound neuromodulation, showing reduced off-target effects and revealing non-auditory side effects at higher pressures.

This study identifies the mechanism by which focused ultrasound excites neurons through mechanosensitive calcium channels and ion channel amplification.