Author

Bruno Lopes-Bastos

Centre National de la Recherche ScientifiqueORCID 0000-0002-0517-9455Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Research Focus:development & morphogenesis· 2 Approach:experimental assay· 2 Organism:zebrafish· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Giulia AllavenaiD· 2 shared Miguel Godinho FerreiraiD· 2 shared Andreia MarquesiD· 1 shared Aurora Irene IdilliiD· 1 shared Eirini TrompoukiiD· 1 shared Fabrizio d’Adda di FagagnaiD· 1 shared Francesca RossielloiD· 1 shared Marina MioneiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersCell Biology

Giulia AllavenaORCID

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2 papers

Recent·Most cited

cGAS-STING is responsible for aging of telomerase deficient zebrafish

Naz Şerifoğlu, Giulia Allavena, Bruno Lopes-Bastos, Marta Marzullo, Andreia Marques, Pauline Colibert, Pavlos Bousounis, Eirini Trompouki, Miguel Godinho Ferreira

This study shows that cGAS-STING contributes to premature aging in telomerase-deficient zebrafish by promoting senescence and inflammation, and its absence rescues aging-related phenotypes.

The EMBO Journal · Jun 9, 2025Peer-reviewedCell Biology

Telomeric antisense oligonucleotides reduce premature aging phenotypes in telomerase mutant zebrafish

Giulia Allavena, Francesca Rossiello, Aurora Irene Idilli, Marina Mione, Fabrizio d’Adda di Fagagna, Miguel Godinho Ferreira, Bruno Lopes-Bastos

This study shows that telomeric antisense oligonucleotides reduce premature aging in telomerase mutant zebrafish by inhibiting DDR signaling.

bioRxiv · May 23, 2025 · PreprintPathology