Author

Michael A. Goodman

University of UtahORCID 0000-0002-0772-3274Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Amav Khambete· 1 shared Ernest M. Kameni· 1 shared Estelle Atiogue· 1 shared Francis Sprouse· 1 shared Hanghang WangiD· 1 shared Hyun Cheol RohiD· 1 shared Jisun SoiD· 1 shared Kimberley Jane EvasoniD· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperPathology

Amav Khambete

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1 paper

Recent·Most cited

Diet-induced MASH liver fibrosis promoted by EphB2 can be targeted by small molecule tetramerization inhibitors

Masaaki Yoshigi, Séverin Donald Kamdem, Hanghang Wang, Amav Khambete, Francis Sprouse, Ernest M. Kameni, Estelle Atiogue, Sarah Pellizzari, Kimberley Jane Evason, My N. Helms, Michael A. Goodman, Shuang Wang, Jisun So, Hyun Cheol Roh, Sihem Boudina, , Patrice Nsangou Mimche

This study identifies EphB2 as a driver of MASH liver fibrosis and shows that targeting EphB2 with small molecule inhibitors can reduce fibrosis in mouse models.

bioRxiv · Nov 19, 2025 · PreprintPathology