Author

Cailin S. Deiter

University of Colorado AnschutzORCID 0000-0002-0772-6387Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Neuroscience· 1 Systems Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Organism:mouse· 2 Approach:statistical model· 2 Assay:metabolomics· 1 Research Focus:protein structure prediction· 1

Frequent Co-Authors

Bobbie‐Jo Webb‐RobertsoniD· 1 shared Carmella Evans‐MolinaiD· 1 shared Carrie NicoraiD· 1 shared Charles K. AnsongiD· 1 shared David W. DonleyiD· 1 shared Decio Laks EizirikiD· 1 shared Dylan SarbaughiD· 1 shared Ernesto Satoshi NakayasuiD· 1 shared

Activity by Year

2019-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 28 mutual co-author links

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1 shared paperSystems Biology

Bobbie‐Jo Webb‐RobertsonORCID

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3 papers

Recent·Most cited

Stromal innervation is prognostic in non-muscle-invasive bladder cancer and predicted by a distinct microenvironmental transcriptomic signature

Cailin S. Deiter, Florus C. de Jong, Mitchell Olislagers, Kimberly Rae Jordan, Tahlita C M Zuiverloon, James C. Costello

The paper identifies a transcriptomic signature predicting stromal innervation in NMIBC and links it to clinical outcomes.

bioRxiv · Sep 23, 2026 · PreprintCancer Biology

The role of islet lipid composition remodeling in regulation of beta-cell death via ADP-ribosyl-acceptor glycohydrolase ARH3 signaling in insulitis

Ernesto Satoshi Nakayasu, Cailin S. Deiter, Jennifer E Kyle, Michelle A. Guney, Dylan Sarbaugh, Ruichuan Yin, Yi Cui, Carrie Nicora, Farooq Syed, Jonas Juan-Mateu, Raghavendra G. Mirmira, Carmella Evans‐Molina, Decio Laks Eizirik, Bobbie‐Jo Webb‐Robertson, Kristin Burnum-Johnson, , Charles K. Ansong

The paper investigates lipid composition changes in insulitis models and identifies ARH3 as a regulator of beta-cell apoptosis through PLA2G6 signaling.

bioRxiv · Mar 23, 2020 · PreprintSystems Biology

Latent Toxoplasma gondii infection increases soluble mutant huntingtin and promotes neurodegeneration in the YAC128 mouse model of Huntington's disease

David W. Donley, Teal Jenkins, Cailin S. Deiter, Reed Campbell, Marley Realing, Vanita Chopra, Stephen Hersch, Jason P. Gigley, Jonathan H. Fox

This study shows that latent Toxoplasma gondii infection worsens neurodegeneration in YAC128 HD mice by increasing soluble mutant huntingtin and neurofilament light protein levels.

bioRxiv · Feb 15, 2019 · PreprintNeuroscience