Vikas Bansal

This study uses snATAC-seq and snRNA-seq to identify TE-derived regulatory elements involved in adult hippocampal neurogenesis in response to rhEPO stimulation.

The paper investigates transcriptomic changes in oligodendroglial lineage cells from PD patient-iPSCs with LRRK2-G2019S mutation, identifying altered pathways and signaling in OPCs and ODCs.

This study identifies shared genetic loci between Parkinson's disease and schizophrenia using GWAS and scRNA-seq, highlighting RAI1's role in mitochondrial dysfunction and non-motor symptoms.

The paper shows that deleting EPO in the forebrain of mice leads to increased expression of EPOR and EphB4, which may explain the lack of cognitive deficits in these mutants.

The paper identifies transcriptomic changes in oligodendrocytes and precursor cells linked to Parkinson's disease outcomes and shows their predictive value for cognitive and depressive symptoms.

The paper presents a multi-layered molecular dataset from iPSC-derived dopaminergic neurons to model Parkinson's disease genetic risk and provides a data browser for accessing these resources.

The paper identifies Err2 and Err3 as transcription factors promoting functional properties in gamma motor neurons essential for proprioceptive movement control.

The paper identifies early translatome changes in specific neural cell types before EEG and neuropathological signs of prion disease in mice.

The paper maps human coronavirus entry factors using single-cell RNA-seq to identify permissive cell types and tissues for SARS-CoV-2 infection.

This paper introduces cscGANs for generating realistic single cell RNA-seq data to augment sparse datasets, improving analysis outcomes and reducing experimental costs.

The paper reanalyzes single-cell transcriptomic data and in situ stains to reveal the presence of a transcriptomically homologous inner cell mass in human blastocysts, highlighting unique developmental and evolutionary features.

This study uses genome-wide association data for educational attainment and schizophrenia to identify genetic heterogeneity and novel candidate genes for schizophrenia.

The paper estimates the extent and biological role of mutually exclusive splicing in humans by identifying and validating over 855 MXEs, including novel exons, and links them to disease pathology.