Vivek V. Thacker

This study demonstrates the use of a lung-on-chip model to study drug vehiculation via pulmonary surfactant, showing effective delivery and suppression of Mycobacterium tuberculosis.

The paper develops a human urothelial organoid model to study urine-urothelium interactions in UTI resilience and UPEC recurrence, finding that high-solute urine weakens tissue and promotes CWD UPEC formation.

This study reveals the structural organization of Mycobacterium tuberculosis biofilms, showing how bacterial cords and an amyloid-like matrix contribute to antibiotic tolerance.

This study shows that altering macrophage metabolism can reduce drug tolerance in Mycobacterium tuberculosis by affecting bacterial redox states.