Ulrich Karl Steiner

This study shows that gene amplification increases antibiotic resistance but also imposes growth costs and increases susceptibility to high antibiotic concentrations.

This study shows that protein aggregation in E. coli drives aging by reducing intracellular space, leading to larger cell sizes and asymmetric divisions.

The paper investigates how single-cell damage dynamics under stress affect population fitness, finding that damage-dependent mortality is a key factor.

The paper shows that division asymmetry in E. coli has evolved to an optimum for population growth, with selective forces primarily acting on transcription signal asymmetry variance.

This study investigates why holometabolous insects evolved a pupal stage, finding that rapid growth and a trade-off between growth and differentiation favor this life history under high mortality.

The paper shows that senescence in Escherichia coli is shaped by two stochastic processes: random cellular deterioration and asymmetric transmission of an unknown factor during cell division.