Author

Jeremy E. Turnbull

University of CopenhagenORCID 0000-0002-1791-754XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Andrew J. RoeiD· 1 shared Andrew J. SmithiD· 1 shared Carla JohnsoniD· 1 shared Cheng‐Hock TohiD· 1 shared Christopher A. MoxoniD· 1 shared Dagmara McGuinnessiD· 1 shared Edwin Alexander YatesiD· 1 shared Fadil A. BidmosiD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperMicrobiology

Andrew J. RoeORCID

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1 paper

Recent·Most cited

Bacterial histone-like proteins released during antibiotic treatment mediate vascular injury in meningococcal sepsis.

Dagmara McGuinness, Carla Johnson, Leandro Lemgruber, Fadil A. Bidmos, Edwin Alexander Yates, Jeremy E. Turnbull, Tom J. Evans, Rebecca E. McHugh, Andrew J. Roe, Gill R. Douce, Andrew J. Smith, Roisin Ure, Cheng‐Hock Toh, Simon Timothy Abrams, Jethro Adam Herberg, , Christopher A. Moxon

Antibiotic treatment of meningococcal and other sepsis causative bacteria triggers the release of histone-like proteins (HLPs) that damage the endothelium and drive coagulopathy, but these effects can be neutralized by non-anticoagulant heparins or anti-HLP antibodies.

bioRxiv · Jul 16, 2026 · PreprintMicrobiology