Author

Alice Marino

UCLouvainORCID 0000-0002-1985-6576Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Physiology· 2 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

Christophe BeauloyeiD· 3 shared Caroline BouziniD· 2 shared Hrag Esfahani· 2 shared Jérôme AmbroiseiD· 2 shared Luc BertrandiD· 2 shared Sandrine HormaniD· 2 shared Andreas Unger· 1 shared Audrey Ginion· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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3 shared papersPhysiology

Christophe BeauloyeORCID

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3 papers

Recent·Most cited

Platelet GARP-dependent activation of TGF-β1 limits inflammation and promotes cardiac repair after myocardial infarction

Cécile Dufeys, Julie Bodart, Audrey Ginion, Jérôme Ambroise, Noah Trusgnach, Estelle L. Ollivier, Caroline Bouzin, Davide Brusa, Camille Michiels, Yotis A. Senis, Zoltán Nagy, Alice Marino, Luc Bertrand, Christophe Beauloye, Sophie Lucas, Sandrine Horman

This study shows that platelet GARP activates TGF-β1 to limit inflammation and promote cardiac repair after myocardial infarction.

bioRxiv · Jul 6, 2026 · PreprintPathology

skNAC is a Key Driver of Cardiomyocyte Integrity Against Pathological Cardiac Hypertrophy and Heart Failure

Laura Guilbert, Justine Dontaine, Natacha Fourny, Ettore Vanni, Michele Russo, Hugo Vanderroost, Jade Dron, Jérôme Ambroise, Hrag Esfahani, Caroline Bouzin, Younès Achouri, Emilie Hendrickx, Nassiba Menghoum, B. Bearzatto, D. Vertommen, , Luc Bertrand

This study identifies skNAC as critical for maintaining cardiomyocyte integrity and preventing heart failure by regulating proteome homeostasis and metabolic adaptation during pathological hypertrophy.

bioRxiv · Oct 23, 2025 · PreprintPhysiology

When the liver is in poor condition, so is the heart - cardiac remodelling in MASH mouse models

Sébastian Bott, Justine Lallement, Alice Marino, Evangelos P. Daskalopoulos, Christophe Beauloye, Hrag Esfahani, Chantal Dessy, Isabelle Anne Leclercq

This study examines cardiac remodelling in MASH mouse models, finding adverse structural changes in the heart linked to liver disease, not overfeeding or aging.

Clinical Science · Aug 29, 2024Peer-reviewedPhysiology