Author

Justine Lallement

UCLouvain

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:mouse· 2 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:metabolomics· 1

Frequent Co-Authors

Chantal DessyiD· 2 shared Kristian SerafimoviD· 2 shared Olivier FéroniD· 2 shared Sultan Al‐SiyabiiD· 2 shared Amorim Cdos SiD· 1 shared Caroline BouziniD· 1 shared Céline Cruciani‐GuglielmacciiD· 1 shared Céline Guilbaud· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersCell Biology

Chantal DessyORCID

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Author profile →

3 papers

Recent·Most cited

Acidosis-triggered fatty acid overload induces endothelial cell dysfunction.

Sultan Al‐Siyabi, Sébastien Ibanez, Kristian Serafimov, Justine Lallement, Dorothée Marchand, Florine Laloux-Morris, Céline Guilbaud, Delphine Demulder, Hanne Vlieghe, Saeid Moghassemi, Caroline Bouzin, Amorim Cdos S, Olivier Féron, Chantal Dessy

This study shows acidosis enhances fatty acid uptake in endothelial cells, leading to lipid droplet formation, ER stress, and reduced nitric oxide, contributing to endothelial dysfunction.

bioRxiv · Jul 10, 2026 · PreprintCell Biology

Hepatic ADMA/PRMT1 axis regulation is associated with NO-dependent endothelial dysfunction in MASH

Justine Lallement, Sébastian Bott, Hrag Esfahani, Kristian Serafimov, Maxence Henderson, Zoé Benoit, Laurent Dumas, Sultan Al‐Siyabi, Olivier Féron, Isabelle Anne Leclercq, Chantal Dessy

This study investigates how MASH impairs endothelial function via NO-dependent mechanisms and identifies ADMA as a key liver-derived mediator.

bioRxiv · Jun 10, 2025 · PreprintPhysiology

Serine palmitoyltransferase-mediated de novo sphingolipid biosynthesis is required for normal insulin production and glucose tolerance

Chloé Amouyal, Shiou Ping Chen, Jessica Denom, Ilyès Raho, Nadim Kassis, Marc Diedisheim, F. Mifsud, Justine Lallement, Eleni Georgiadou, Mark R. Ibberson, Mikaël Croyal, Xian C Jiang, Céline Cruciani‐Guglielmacci, Hervé Le Stunff, Guy A. Rutter, Chr̀istophe Magnan

This study shows that de novo ceramide synthesis in beta-cells is essential for normal insulin production and glucose tolerance, as its inhibition leads to reduced beta-cell mass and impaired glucose regulation.

bioRxiv · May 14, 2025 · PreprintPhysiology