Author

Puneet Bagga

St. Jude Children's Research HospitalORCID 0000-0002-2264-2341Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Bioengineering· 1 Cancer Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Assay:imaging· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1

Frequent Co-Authors

Ritambhar BurmaniD· 3 shared Kasturee ChakrabortyiD· 2 shared Kirsten K. NessiD· 2 shared Alaa RefaatiD· 1 shared Anang A. ShelatiD· 1 shared Ankhbayar Lkhagva· 1 shared Anthony A. HighiD· 1 shared Areeb Khan· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 26 mutual co-author links

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3 shared papersNeuroscience

Ritambhar BurmanORCID

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3 papers

Recent·Most cited

Neurometabolic correlates of accelerated aging and neurocognitive late effects in long-term survivors of pediatric hodgkin lymphoma and acute lymphoblastic leukemia

Kyla David Gibney, Areeb Khan, Sabah Nisar, Kasturee Chakraborty, Ritambhar Burman, Pat Hanby, Stephanie L. Guthrie, Brian Potter, Melissa Maria Hudson, Kirsten K. Ness, Tara M. Brinkman, Belinda Neal Mandrell, Cai Li, Kevin R. Krull, Puneet Bagga

The paper examines neurometabolic correlates of accelerated aging and neurocognitive late effects in adult survivors of pediatric cancers using 1H MRS and cognitive tests.

bioRxiv · Feb 18, 2026 · PreprintNeuroscience

A novel CEST-based approach for reliably assessing skeletal muscle oxidative phosphorylation: OXCEST

Ritambhar Burman, Yuxi Pang, Asim Kumar Bag, Richard S. Finkel, Kirsten K. Ness, Puneet Bagga

This paper introduces OXCEST, a CEST MRI method to assess skeletal muscle oxidative phosphorylation, showing strong correlation with 31P-MRS measurements.

Magnetic Resonance in Medicine · Oct 22, 2025Peer-reviewedBioengineering

SLFN11 Loss-Induced Chemoresistance is Associated with Overexpression of Glycerophospholipid Biosynthesis in Ewing Sarcoma.

Kasturee Chakraborty, Ritambhar Burman, Saharsh Satheesh, Matthew Kieffer, Chandni Karuhatty, Zuo‐Fei Yuan, Haiyan Tan, Ankhbayar Lkhagva, Anthony A. High, Xusheng Wang, Alaa Refaat, Weixing Zhang, Yaxu Wang, Yiping Fan, M. Madan Babu, , Puneet Bagga

The paper identifies glycerophospholipid biosynthesis upregulation as a mechanism of chemoresistance in SLFN11-loss Ewing sarcoma and shows that inhibiting this pathway enhances DNA-damaging agent efficacy.

bioRxiv · May 9, 2025 · PreprintCancer Biology