Georges M. G. M. Verjans

This study shows HSV infection increases ALS risk and promotes TDP-43 pathology through ICP0-mediated disruption of PML bodies.

This paper establishes HD10.6 neurons as an in vitro model for studying VZV latency and reactivation, showing they support viral infection, persistence, and reactivation with capsaicin.

This study identifies interspecies recombination events in HSV-2 genomes and shows their impact on genetic variation and T-cell recognition.

The paper identifies and characterizes a unique VZV latency transcript (VLT) that represses the viral transactivator gene ORF61 during latency, unifying VZV latency with other herpesviruses.