Author

Simon Andersson

University of HelsinkiORCID 0000-0002-2688-2318Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:development & morphogenesis· 2 Organism:Drosophila· 2 Approach:genetic perturbation / CRISPR· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Pekka KatajistoiD· 3 shared Tuuli KirkineniD· 2 shared Ville HietakangasiD· 2 shared Alessandro Ori‬‬iD· 1 shared Alexandra RitchieiD· 1 shared Anna WebbiD· 1 shared Anne JuutiiD· 1 shared Ari P. RistimakiiD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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3 shared papersPhysiology

Pekka KatajistoORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

PhysiologybioRxivPreprintNov 4, 2026

Nutrient-responsive midgut turnover is spatially controlled by serotonin signaling

Nicole Lamichane, Petteri Borgman, Terhi Rintakangas…·Ville Hietakangas

The paper shows serotonin signaling spatially controls nutrient-responsive midgut turnover in Drosophila by regulating enteroblast maturation and morphological changes through the 5HT7 receptor.

DOI 10.64898/2026.11.03.763885

Drosophilaproteomics

𝕏 PostBluesky

PhysiologybioRxivPreprintNov 4, 2026

mTORC1-dependent control of Hedgehog signaling mediates nutrient-responsive licensing of intestinal stem cell differentiation

Gaia Fabris, Simon Biaudet, Atte Hallasaari…·Ville Hietakangas

This study identifies an mTORC1-PWP1-Hedgehog circuit that links nutrient metabolism to intestinal stem cell differentiation in Drosophila, showing its role in aging and tissue renewal.

DOI 10.64898/2026.10.29.762847

Drosophilagenetic perturbation / CRISPR

𝕏 PostBluesky

Developmental BiologyCell stem cellPeer-reviewedMar 6, 2025

Fetal-like reversion in the regenerating intestine is regulated by mesenchymal Asporin

Sharif Iqbal, Simon Andersson, Ernesta Nesta…·Pekka Katajisto

The paper shows that mesenchymal Asporin mediates fetal-like reprogramming in regenerating intestinal epithelium by modulating Tgfβ signaling through CD44.

24 citationstop 1%

mousebulk RNA-seq

𝕏 PostBluesky