Author

Franz Oswald

Universität UlmORCID 0000-0002-2923-5765Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:differential expression· 1 Organism:Drosophila· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Organism:mouse· 1 Research Focus:protein structure prediction· 1 Approach:statistical model· 1

Frequent Co-Authors

Aleksandra Turkiewicz· 1 shared Amy Schug· 1 shared Brian A. GebeleiniD· 1 shared Ellen K. GaglianiiD· 1 shared Emily Rose Harding-TheobaldiD· 1 shared Karin Scharffetter‐KochanekiD· 1 shared Lisa M. GutzwilleriD· 1 shared M. Wagner· 1 shared

Activity by Year

2022-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperGenetics

Aleksandra Turkiewicz

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2 papers

Recent·Most cited

ER-stress signaling and Alzheimer's proteins adjust the quality of human protein synthesis

Zhouli Cao, Max Hartmann, M. Wagner, Amy Schug, Reinhild Roesler, Sebastian Wiese, Qingwen Yang, Franz Oswald, Karin Scharffetter‐Kochanek, Sebastian Iben

The paper shows that ribosomal error-rate adapts in human aging models but not in mouse models, involving ER-stress signaling and Alzheimer's proteins.

bioRxiv · Feb 24, 2026 · PreprintCell Biology

A Drosophila Su(H) Model of Adams-Oliver Syndrome Reveals Notch Cofactor Titration as a Mechanism Underlying Developmental Defects

Ellen K. Gagliani, Lisa M. Gutzwiller, Yi Kuang, Yoshinobu Odaka, Phillipp Hoffmeister, Stefanie Hauff, Aleksandra Turkiewicz, Emily Rose Harding-Theobald, Patrick J. Dolph, Tilman Borggrefe, Franz Oswald, Brian A. Gebelein, Rhett A. Kovall

This study uses a Drosophila Su(H) model to show that AOS-linked RBPJ variants cause developmental defects by sequestering Notch cofactors, leading to dysregulated target gene expression.

PLoS Genetics · Aug 11, 2022Peer-reviewedGenetics