L. Miguel Martins

This study shows that developmental mitochondrial complex I dysfunction reduces lifespan and stress resistance, while adult-onset dysfunction increases lifespan and stress resistance.

The paper links amyloid-β forms to sleep duration changes in Alzheimer's disease via NAD+-linked redox metabolism using Drosophila models and human data.

This study shows that developmental mitochondrial dysfunction limits lifespan in Drosophila, while adult dysfunction accelerates ageing, with metabolic adaptations during development being critical for survival.

The paper investigates the role of WDPCP in alcohol consumption, lipid metabolism, and liver cirrhosis using human and model organism studies.

The paper shows that increasing mitochondria-ER contacts in Drosophila improves locomotion, extends lifespan, and reduces Alzheimer's disease symptoms.