Author

Vilma Lammi

University of HelsinkiORCID 0000-0002-3256-5239Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:GWAS summary statistics· 3 Organism:human· 3 Approach:statistical model· 3 Research Focus:GWAS / association mapping· 2 Approach:experimental assay· 1 Research Focus:fine-mapping· 1

Frequent Co-Authors

Beatriz Guillén‐GuíoiD· 2 shared Hanna Maria OllilaiD· 2 shared Jacqueline M. LaneiD· 2 shared Nasa Sinnott-ArmstrongiD· 2 shared Richa SaxenaiD· 2 shared Samuel E. JonesiD· 2 shared Tomoko NakanishiiD· 2 shared Aarno V. PalotieiD· 1 shared

Activity by Year

2023-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersGenetic & Genomic Medicine

Beatriz Guillén‐GuíoORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Human genetics implicate thromboembolism in the pathogenesis of long COVID in individuals of European ancestry

Art Schuermans, Andreas Verstraete, Vilma Lammi, Tomoko Nakanishi, Maddalena Ardissino, Jef Van den Eynde, Benjamin B. Sun, Marios K. Georgakis, Beatriz Guillén‐Guío, Louise V. Wain, Christopher Edward Brightling, PHOSP-COVID Collaborative Group, Johan Van Weyenbergh, Adam J. Lewandowski, Betty Raman, , Peter B. Verhamme

This study identifies a genetic association between thromboembolism risk and long COVID in individuals of European ancestry, suggesting thromboembolism as a potential contributor to long COVID pathogenesis.

Nature Cardiovascular Research · Dec 16, 2025Peer-reviewedGenetic & Genomic Medicine

Genome-wide Association Study of Long COVID

Vilma Lammi, Tomoko Nakanishi, Samuel E. Jones, Shea J. Andrews, Juha M Karjalainen, Beatriz Cortés, Heath O’Brien, Ana Ochoa-Guzmán, Brian Fulton‐Howard, Martin E. Broberg, Hele Haapaniemi, Masahiro Kanai, Matti Pirinen, Axel Schmidt, Ruth E. Mitchell, , Sergi Papiol

This study performs a genome-wide association study for Long COVID, identifying a significant association with the FOXP4 locus and highlighting the role of pulmonary dysfunction and COVID-19 severity.

Nature Genetics · May 21, 2025Peer-reviewedGenetic & Genomic Medicine

The alpha-2A-adrenergic receptor (ADRA2A) modulates susceptibility to Raynaud's syndrome

Anniina M. Tervi, Markus O Ramste, Erik Abner, Paul P. Cheng, Jacqueline M. Lane, Matthew Maher, Vilma Lammi, Satu J. Strausz, Trieu Nguyen, Mauro Lago‐Docampo, Wenduo Gu, Tõnu Esko, Richa Saxena, Aarno V. Palotie, Samuli Ripatti, , Hanna Maria Ollila

This study identifies ADRA2A as a major risk locus for Raynaud's syndrome through genome-wide association analysis and functional validation in vascular smooth muscle cells.

medRxiv · Oct 4, 2023 · PreprintGenetic & Genomic Medicine