Author

Samantha Katarzyna Dziurdzik

University of British ColumbiaORCID 0000-0002-3553-1595Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 2 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Organism:yeast· 3 Research Focus:chromatin & epigenetic regulation· 2 Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Research Focus:metabolism & bioenergetics· 1 Approach:statistical model· 1

Frequent Co-Authors

Elizabeth ConibeariD· 3 shared Michael DaveyiD· 3 shared Arash BashirullahiD· 1 shared Björn D. M. BeaniD· 1 shared Hailey EngiD· 1 shared Jordan Faith DrotskyiD· 1 shared Junran YaniD· 1 shared Kevin Ryan JeffersiD· 1 shared

Activity by Year

2020-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 28 mutual co-author links

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3 shared papersCell Biology

Elizabeth ConibearORCID

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3 papers

Recent·Most cited

A shared binding interface controls Vps13 organelle-specific targeting independently of its vacuolar protein sorting function

Kevin Ryan Jeffers, Samantha Katarzyna Dziurdzik, Michael Davey, Jordan Faith Drotsky, Elizabeth Conibear

The paper shows that Vps13 binds adaptors via a shared PxP motif interface, but this interface is not required for its vacuolar protein sorting function.

Molecular Biology of the Cell · Jan 15, 2026Peer-reviewedCell Biology

Hoi1 targets the yeast BLTP2 protein to ER-PM contact sites to regulate lipid homeostasis

Samantha Katarzyna Dziurdzik, Vaishnavi Sridhar, Hailey Eng, Sarah D. Neuman, Junran Yan, Michael Davey, Stefan Taubert, Arash Bashirullah, Elizabeth Conibear

This study identifies Hoi1 as an adaptor that targets BLTP2-like proteins to ER-PM contact sites in yeast, regulating lipid homeostasis and showing conservation in worms and flies.

bioRxiv · Feb 11, 2025 · PreprintCell Biology

A VPS13D spastic ataxia mutation disrupts the conserved adaptor binding site in yeast Vps13

Samantha Katarzyna Dziurdzik, Björn D. M. Bean, Michael Davey, Elizabeth Conibear

The paper identifies a disease-causing mutation in VPS13D that disrupts adaptor binding and membrane recruitment in yeast, supporting a conserved role for the VAB domain.

Human Molecular Genetics · Jan 10, 2020Peer-reviewedMolecular Biology