Author

Irene B. Maroto

Universidad Complutense de MadridORCID 0000-0002-4156-166XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:electronic health records· 1 Approach:statistical model· 1

Frequent Co-Authors

Abel Eraso‐PichotiD· 1 shared Alba Fernández‐RodrigoiD· 1 shared Alba Hermoso-López· 1 shared Albert QuintanaiD· 1 shared Alicia Álvaro-BlázqueziD· 1 shared Andrea Sánchez-RuiziD· 1 shared Antonio Muñoz‐Callejas· 1 shared Carla Sánchez‐CastilloiD· 1 shared

Activity by Year

2021-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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1 shared paperNeuroscience

Abel Eraso‐PichotORCID

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3 papers

Recent·Most cited

Astrocytic PI3Kα controls synaptic plasticity and cognitive function via serine metabolism

Alba Fernández‐Rodrigo, Celia García Vilela, Abel Eraso‐Pichot, Irene B. Maroto, María D. G. Moreno, Esperanza López-Merino, Silvia Gutiérrez-Eisman, Carla Sánchez‐Castillo, Cristina Boers-Escuder, Eneko Merino-Casamayor, Mariona Graupera, Albert Quintana, María Isabel Cuartero, Maite Solas, Giovanni Marsicano, Jose Antonio Esteban

This study shows that astrocytic PI3Kα supports synaptic plasticity and cognitive function by regulating serine metabolism and D-serine levels necessary for NMDA receptor activation during long-term potentiation.

Nature Communications · Sep 4, 2026Peer-reviewedNeuroscience

The CB1 receptor interacts with cereblon and drives cereblon deficiency-associated memory shortfalls

Carlos Costas‐Insua, Alba Hermoso-López, Estefanía Moreno, Carlos Montero-Fernández, Alicia Álvaro-Blázquez, Irene B. Maroto, Andrea Sánchez-Ruiz, Rebeca Dı́ez-Alarcia, Cristina Blázquez, Paula Morales, Enric I. Canela, Vicent Casadó, Leyre Urigüen, Gertrudis Perea, Luigi Bellocchio, , Manuel Guzmán

This study identifies CB1R overactivation as a driver of CRBN deficiency-linked ARNSID and suggests CB1R blockade as a potential therapy.

EMBO Molecular Medicine · Mar 21, 2024Peer-reviewedNeuroscience

IGF-1 facilitates extinction of conditioned fear

Jose Antonio Noriega‐Prieto, Laura Eva Maglio, Irene B. Maroto, Jesús Martín-Cortecero, Antonio Muñoz‐Callejas, Marta Callejo-Móstoles, David Fernández de Sevilla

This study shows IGF-1 enhances fear extinction memory by modulating synaptic plasticity and neuronal excitability in the infralimbic cortex.

eLife · Apr 1, 2021Peer-reviewedNeuroscience